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Importantly, our cell culture observations were corroborated by our results obtained in animal studies, suggesting that increased hepatic protein S -glutathionylation contributes to NAFLD pathogenesis via suppression of the NF-B signaling pathway in hepatocytes
Carbonyl compounds in the proteins were quantified by precipitating proteins with 20% trichloroacetic acid, followed by resuspension in dinitrophenylhydrazine (DNPH)
Glutathione may help shift the production from the darker pigment to the lighter one
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